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Herein, the action of melatonin on various GI cancers, with the emphasis on its effect on major oncogenic signaling pathways (e.g., Wnt/-catenin signaling pathway, phosphatidylinositol 3-kinase/protein kinase B (PI3K/Akt), and nuclear factor kappa B (NF-B)) are discussed
The theoretical concern remains exactly that
Some preclinical studies suggest GLP-1 signaling may have anti-inflammatory potential through intestinal barrier protection, while others indicate that rapid changes in gut motility could stress an already inflamed mucosa
Promising results but caution needed in GLP-1RA large scale epidemiology
NAFLD is associated with metabolic syndromes and the incidence of NAFLD has increased over time [4, 5]
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