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It is, therefore, concluded that GSH supplementation could not increase the small intestinal mucosal GSH level of weaned piglets, and this could potentially relate to the kinetics of GSH in the digestive tract, where GSH seemed to be prone to the breakdown to CYS and CYSS and the auto-oxidation to GSSG

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WI-38 fibroblasts were exposed to lipopolysaccharide (LPS) in vitro, while an animal model of pneumonia was generated through intratracheal LPS administration in mice

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