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salina 99 Ds GPDH 699 variant, they used for structural studies, had a functional N-terminal GPP domain 24 we think that it was truncated (this explains why our 98 Ds GPDH 701 construct when expressed in HeLa cells accumulated high levels of Gro3P and had a profound proliferation defect) ( Figure 1d, h

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Valizadeh M, Ahmadzadeh A, Behzadi M, Yeganeh F

This is significant because of the change in cellular redox state found in both diabetes and AD

Consequently, the response of brain tissue to radiation may differ between rodent preclinical models and human patients, particularly at the molecular level