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In the hippocampal CA1 region, VPA-exposed rats showed significant SV depletion compared to controls ( P 0.05 )

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Conformational analysis by nuclear magnetic resonance (NMR) spectroscopy indicated that the observed state is distinct from apo, ADP- or slowly hydrolysable ATPS states

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Mito-TEMPO, a mitochondrion-targeted antioxidant, significantly rescued doxorubicin (DOX)-induced myocardial ferroptosis, substantiated that mitochondrial oxidative damage underlies ferroptosis-induced cardiac injury [57]