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Caveolin-1 mutations (P132L and null) and the pathogenesis of breast cancer: caveolin-1 (P132L) behaves in a dominant-negative manner and caveolin-1 (-/-) null mice show mammary epithelial cell hyperplasia

Bacterial burden was subsequently quantified by manual colony counting, with results expressed as CFU per organ

These data indicate that PAPR-1 induces LXR poly(ADP-ribosyl)ation, therefore inhibiting LXR-induced ABCA1 expression in INS-1 cells