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The influence of hypoxia on the differentiation of MSCs has been a subject of debate, with studies reporting a spectrum of outcomes, including inhibition (Yang et al., 2011

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Caspase-mediated loss of mitochondrial function and generation of reactive oxygen species during apoptosis

TdGSH erased LOsG-induced cell metabolic memory (MM) in ROS homeostasis Glucose/ROS pathways promote cellular adaptive processes, which can alleviate glucose-dependent ROS stress-induced detrimental effects on cell survival and subsequently allows for the development of MM 18 , which exists in diabetic patients