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Mechanically, Palmitate enhances the binding of LPS to TLR4, thereby promoting TLR4 dimer formation and endocytosis for subsequent activation of the PI3K/Akt and NF-B pathways and bacteria phagocytosis (88, 89)

Altered functional B cell subset populations in patients with chronic fatigue syndrome compared to healthy controls

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Importantly, the excellent biocompatibility and minimal toxicity of PDA@Pt render it a highly promising uric-acid-lowering and anti-inflammatory agent for the treatment of acute gout

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