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Future research is required to examine the extent to which folate-related epistatic interactions respond to environmental cues, such as UVR exposure

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and deepening mechanistic investigations to clarify direct mechanisms of incretin receptor signalling in fibroblasts and immune cells across different tissue types

LEO alleviates T2DM by remodeling the gut microbiota ecosystem, enhancing butyrate biosynthesis, and activating an MPC2-dependent mitochondrial energy metabolism pathway to reverse GLP-1 secretory dysfunction in intestinal L cells