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aeruginosa , the MexR repressor senses oxidative stress (two Cys residues form disulfides), dissociates from the mexAB-oprM promoter, and derepresses efflux under peroxide stress

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Luo M, Zheng L-W, Wang Y-S, Huang J-C, Yang Z-Q, Yue Z-P, et al

Therapeutically, targeting key mediators (anti-TNF strategies, IL-6 signalling modulators, or chemokine receptor blockers) or may contribute to (gut-derived LPS, dysbiosis) has the potential to disrupt peripheral-to-central inflammatory transmission, these provided interventions are timed and tailored to the patients immune profile [98]

In reaction to AD, astrocytes experience a shift in profile, one that is reactive and pro-inflammatory while losing homeostatic roles thus disrupting BBB integrity, ion and neurotransmitter buffering, and energy metabolism (Liddelow et al., 2017