glp-1 drugs and alzheimer's Blockbuster Diabetes Weight Loss Fails in Trial What's Next for Alzheimer's Treatment?

Jamwal et al., 2017)

Al Rajabi A 1 , Castro GS, da Silva RP, Nelson RC, Thiesen A, Vannucchi H, Vine DF, Proctor SD, Field CJ, Curtis JM, Jacobs RL

This could be reversed by overexpression of HK2 induced by plasmid transfection, suggesting that inhibition of HK2 represents a new therapeutic option for (liver) fibrosis (207)

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This is most likely due to the stimulation of the early growth response-1 (egr-1) gene and its co-repressor nerve growth factor 1-A binding protein-2 (naB2), which are also responsible for cytokine and growth factor generation and thereby, early extracellular matrix (collagen) and blood vessel formation (55)