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Abstract Acetaminophen (APAP)-induced acute liver injury (AILI) is primarily driven by CYP3A4mediated overproduction of the toxic metabolite N-acetyl-p-benzoquinone imine (NAPQI), CYP3A4 activity serves as the rate-limiting determinant of NAPQI accumulation levels

Moreover, it should be noted that, specifically, glutathione does not have any health claims authorised by the EFSA (European Union) or by the FDA (United States)

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Chen B, Wei J, Wang W, Cui G, Zhao Y, Zhu X, Zhu M, Guo W, Yu J