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Intracellular replication has been found to be supported by TonB-dependent iron acquisition from a host source, such as ferritin, the degradation of which is promoted by the pathogen (Larson et al., 2002, Larson et al., 2004)

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Zheng J, et al

However, preclinical studies have demonstrated more promising effects of NAC, including improved systemic and hepatic lipid metabolism, reduced inflammation-induced liver damage, and enhanced glucose tolerance in NAFLD-induced mice, likely due to its role in restoring hepatic glutathione and glutathione reductase levels

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