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Feedback suppression of meal-induced glucagon-like peptide-1 (GLP-1) secretion mediated through elevations in intact GLP-1 caused by dipeptidyl peptidase-4 inhibition: A randomized, prospective comparison of sitagliptin and vildagliptin treatment

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This was made possible by co-formulating semaglutide with the absorption enhancer sodium N-(8-(2-hydroxybenzoyl)amino)caprylate, better known as SNAC

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Conclusions In this review, we have focused on three aspects: the reactivity of FRs, their cellular effects, and the potential use of antioxidants as scavengers of FRs and their role in cellular metabolism