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Abstract In the last few decades, glucagon-like peptide-1 receptor (GLP-1R) agonists have changed current guidelines and improved outcomes for individuals with type 2 diabetes

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14 Transcriptional profiling also revealed that aged MuSCs acquire intrinsic defects in self-renewal capacity, linked to dysregulated p38/ mitogen-activated protein kinase (MAPK) signaling and impaired fibroblast growth factor receptor 1 (FGFR1) activity, changes that are not rescued by a young environment

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