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As the disease progresses, the abnormal synovium invades surrounding tissues, and produces chemical substances that destroy the cartilage and bone of the joint surface

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Importantly, either genetic silencing of NCOA4 or treatment with the iron chelator deferoxamine (DFO) significantly attenuated these pathological changes, resulting in over 60% improvement in neuronal survival (Plascencia-Villa and Perry, 2021)