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In mouse models of Alzheimers disease, triple agonists play a neuroprotective role and ameliorate cognitive deficits by significantly reducing hippocampal amyloid-, phosphorylated tau aggregates, and neuroinflammation, and stimulating neurogenesis and brain-derived neurotrophic factor expression (Li et al., 2018

Remarkably, this inflammation-targeting SP@M restored osteoimmunology homeostasis, inhibited the activation of osteoclasts, protected bone formation, and promoted cartilage recovery in CIA mouse models in vivo (Fig