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By Karin Elgar Abstract Abstract Vitamin B12 (B12) is an essential cofactor in cellular metabolism adequate supplies are needed for normal blood formation and neurological function, and deficiency can therefore lead to macrocytic anaemia and neurological deficits

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Probably due to the lack of complementary interacting residues, superimposing GIP or GLP-1 to GCG bound to GCGR shows that the aligned peptides reduced favorable contacts with GCGR compared to GCG by removing one hydrogen bond (Y10 P (GCG)/Y10 P (GIP)/V16 P (GLP-1)-Q142 1.40b (GCGR)) and pi-pi stacking (Y13 P (GCG)/A13 P (GIP)/Y19 P (GLP-1)-Y138 1.36b (GCGR)) and by repulsing the interaction between Y1 P (GIP) and I235 3.40b (GCGR)