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While essential for heart and vessel function, defective or excessive autophagy can lead to major cardiovascular disorders such as heart failure and atherosclerosis [105]
The albumin-binding C18 di-acid sidechain prolonged the elimination half-life to approximately 30 h in rats and 60 h in monkeys, which is roughly 10-fold longer than native GLP-1, while absolute bioavailability exceeded 85% [42]
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In heart failure patients, greater than 50% increases in BUN were observed in 16.6% of valsartan-treated patients compared to 6.3% of placebo-treated patients [see Warnings and Precautions (5.4)]
