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Retrieved from Pinto, M., Brennan, L., Diehl, K., et al

Recent findings suggest that many of the memory enhancing effects originally attributed to AngII acting at the AT1 receptor subtype were due to the conversion of AngII to angiotensin III (AngIII) and then to the hexapeptide angiotensin IV (Ang IV) and it were AngIV activation of the AT4 receptor subtype that facilitated memory consolidation and retrieval [62-64]

Although it has been reported that the risk of EWDR is increased by faster or more effective lowering of HbA 1c in individuals treated with insulin or a sulfonylurea [14, 25], in our study reduction in HbA 1c was not independently associated with increased odds of the PDR endpoint