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Enhanced J-protein interaction and compromised protein stability of mtHsp70 variants lead to mitochondrial dysfunction in Parkinsons disease

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Loss of both bromine ions is followed by the formation of a highly reactive episulfonium ion which acts as a DNA adduct which is believed to be the root of the compounds tumourigenicity and mutagenicity (Figure 4)

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Modification of Keap1 Cysteine Residues by Sulforaphane