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Actin Sequestration and Cytoskeletal Regulation The principal mechanism of TB-500 involves binding to the N-terminus of G-actin monomers through the LKKTET hexapeptide motif, forming a 1:1 non-covalent complex that sequesters G-actin from the polymerization-competent pool

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BPC-157 is generally associated with localized tissue support, while TB-500 is discussed in relation to broader soft tissue and mobility considerations

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