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Receptor trafficking involves the internalisation of the active receptor: Ligand complex towards endocytic compartments, followed by its deactivation/dephosphorylation and recycling back to the cell membrane, leading to its re-sensitisation, or trafficking to lysosomal organelles for its degradation and final signal termination (McGlone et al., 2021)
The metabolic effects of AKI within the kidney are well-described: disruption of mitochondrial structure, dynamics, biogenesis, and basic electron transport chain function are all present 36
DES has the opposite a tripeptide deletion at the N-terminus that also reduces IGFBP binding but with different receptor-affinity profile and shorter half-life
USA 101 , 1014910154 (2004)
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