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High-throughput screening identified a small TERT-activating compound that could increase TERT transcription in senescent cells, restore it to the physiological expression level in young cells, promote telomere synthesis, reduce DNA damage signals at telomeres, and thus reverse cellular senescence

2: The volume of plasma (blood cells) is diluted by one time

[1] GHK-Cu acts as a potent attractant for various cells involved in the healing process, including capillary cells, mast cells and macrophages

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