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Mechanism investigation in regulatory effects of catalpol on osteoclastogenesis and osteoclast-derived bone loss

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A study in transgenic animals demonstrates that selectively restoring pancreatic GLP-1Rs in GLP-1R null mice normalizes glucose tolerance in these animals suggesting that GLP-1R expression in the pancreas is sufficient to maintain normal glucose metabolism (Lamont et al., 2012)

Postprandial glucose improves the risk prediction of cardiovascular death beyond the metabolic syndrome in the nondiabetic population