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Second, most clinical trials assessing GLP-1 receptor agonists in liver diseases have relatively short follow-up periods, resulting in limited evidence regarding long-term hepatic outcomes

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Concurrently, in insulin-resistant states, increased circulating levels of insulin stimulate hepatic lipogenic enzymes and increase de novo hepatic lipogenesis, exacerbating hepatic steatosis, inflammation, and fibrosis [6, 12]

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Therefore, the CpG island contributes to DMF-mediated CBS regulation in an NRF2-independent manner