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This occurs through noncatalytic stimulation by cytosolic cis-aconitate decarboxylase 1, which is upregulated in response to mycobacterial infection (Yang et al., 2025)

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Such counteraction of encephalopathies in terms of possible ubiquitous neurotransmitter role (i.e., the exogenous effects and endogenous presence to verify endogenous presence and significance) includes the distinctive noxious agents, specifically affecting all brain structures (and consequently neurotransmitter disabilities) with the areas most affected by lesions most presented

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