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Whether that data materially changes clinical decision-making for any individual patient isn't something this article can verify independently

This event was, however, not necessarily preceded by an oxidative burst and later on we demonstrated that GSH release was the result of an active process that the cells carried out through specific carriers in order to efficiently execute the apoptotic program (Ghibelli et al., 1998)

This mechanism is critical in acetaminophen overdose, where the toxic metabolite NAPQI depletes hepatic glutathione N-acetylcysteine administration replenishes GSH and prevents liver failure

(2001) Impaired incretin response after a mixed meal is associated with insulin resistance in nondiabetic men

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