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Preprint at medRxiv (2022)

Although predicted not to be pathogenic using the bioinformatics tool PolyPhen-2, the PSPH V116I mutation is located at the inner core of the PSPH hydrolase domain

specifically tested the hypothesis of a protective effect of G6PD deficiency on the development of postischemic heart failure in a mouse genetically deficient in G6PD [94]