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The principle lipase implicated in TBI is cytoplasmic phospholipase A2 (cPLA2), which is upregulated after in vivo and in vitro experimental TBI and preferentially cleaves arachidonic acid as an essential part of enzymatic lipid peroxidation and inflammatory signal transduction [82]

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In contrast, the ZVAD co-treatment failed to suppress the uptake of PI in RSL3-treated 3D PTC spheroids, mirroring tumor cell viability findings in 2D

Structures of a platelet-derived growth factor/propeptide complex and a platelet-derived growth factor/receptor complex

This reaction results in the temporary formation of oxidized methylcobalamin (III)