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Therefore, we adopted LCA (150 mg/kgBW/d) to facilitate FAO, and further examined the AF vulnerability and atrial remodeling in vivo (a mice model of high-fat diet (HFD)-induced obesity-mediated AF) and in vitro (a primary atrial cardiomyocyte cell model of palmitate (PA)-mimicked lipid overloading), aiming to determine whether enhancing FAO can alter the process that underlie AF in obesity and explore possible mechanisms

Seizure 20 , 369375 (2011)

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