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Such alterations were also associated with inhibition of cytochrome c oxidase (CcOX) activity and microtubule network disassembly, which are considered hallmarks of nitric oxide (NO) toxicity, indicating that NO, rather than the depletion of GSH per se, is the primary mediator of cell damage

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Traumatic brain injury (TBI) support Dihexas synaptogenic and neuroprotective mechanisms make it a candidate of interest for TBI recovery research

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