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In murine models of metabolic syndrome and diet-induced obesity, SLU-PP-332 reduces fat mass, improves lipid parameters, and remodels liver steatosis signatures while shifting respiratory exchange ratio toward greater lipid use

Poly IC triggers a cathepsin D- and IPS-1-dependent pathway to enhance cytokine production and mediate dendritic cell necroptosis

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Conclusion The findings offer new insights into the metabolic pathophysiology of fibroids, which may aid in the development of targeted therapeutic strategies for this widespread gynecological disorder

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