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Exendin-4 has been shown to reduce advanced glycation end product (AGE)-induced interleukin (IL)-6 and TNF- production, the expression of receptor for AGE (RAGE), and cell death in mesangial cells (Wang et al., 2014)

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Here, we show that PKC inhibition with calphostin C markedly reduces triagonist-induced GSIS enhancement in WT islets

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