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Animal models and reviews of real-world data have previously suggested that GLP-1 drugs might reduce the risk or slow development of Alzheimers

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(2000) identified a C-terminal RSRR retention motif in GABA B R1 that retains the receptor on the endoplasmic reticulum, thus preventing surface expression

This leads to dose-dependent weight loss (typically 8-15% in monotherapy trials), with additive effects when combined with GLP-1 agonists like semaglutide due to complementary mechanisms on energy balance and gastrointestinal function

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