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GHK-Cu is studied in laboratory models involving copper peptide signaling, extracellular matrix organization, collagen-associated pathways, tissue-response biology, and cellular repair-associated signaling under controlled experimental conditions

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For example, the inhibition of cell proliferation by curcumin has been associated with the Nrf2-dependent downregulation of DNA repair-specific flap endonuclease 1 (Fen1) in breast cancer cells in culture (46)

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