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In an iron-deficient or hypoxic environment, activation of the SAPK/JNK pathway can enhance transferrin synthesis and iron ion uptake, leading to elevated intracellular iron levels

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ETBF downregulates miR-149-3p in host cells, which results in the upregulation of PHF5A to modulate the alternative splicing of KAT2A, thereby increasing the expression of genes such as SOD2 and promoting intestinal inflammation 122

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Newland et al., 1989