In addition, clinical trials showed that increasing the phagocytic ability, nitric oxide (NO) secretion, and inducible nitric oxide synthase (iNOS) production, this dramatically reduced the proliferation of HepG-2 (Hepatocellular Carcinoma G2) cells [56]
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Modulated expression of 4,000+ human genes in cultured fibroblasts Upregulated antioxidant genes including SOD and glutathione peroxidase Downregulated pro-inflammatory and tissue-destructive gene networks Regenerative and protective actions of the GHK-Cu peptide in the light of the new gene data2014Gene Expression Analysis using the Broad Institute Connectivity Map reveals GHK-Cus gene expression signature overlaps with tissue-remodeling, anti-cancer, and neuroprotective pathways, extending its potential research applications
Quick Reference: Standard Protocol Topical alternative: 0.1-1% concentration applied daily/nightly on a continuous basis
Researchers note that a glutathione supplement is not meant to replace standard chemotherapy treatment for cancer, but it is an antioxidant that needs more study for its potential anticancer effects
![Mechanism of action of GLP-1 [25]](https://www.researchgate.net/publication/256289222/figure/fig1/AS:614177232142337@1523442671231/Mechanism-of-action-of-GLP-1-25.png)
