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M.CassadyJ

Ent1 (GYTLIDL), Ent2 (GVSLIDL), Ent5.1 (IPDLIDLDD), Yap1801 (NLNLIDM), Yap1802 (NPNLIDI) and Apl2.1 (SQDLLDLF)

Adding more of a peptide that is overexpressed in your plaques and helps trigger the autoimmune response is the opposite of what the disease needs, and it carries a real theoretical risk of making things worse

Simply having a COVID-19 infection even if a person was fully recovered from it produced dramatic and quite familiar changes

Furthermore, a comprehensive review published in the Journal of Translational Medicine proposed a unifying model for ME/CFS and Long COVID, highlighting how viral infections (like SARS-CoV-2 or Epstein-Barr Virus) trigger a cascade of immune exhaustion, micro-clotting, and severe oxidative stress