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In chronic or biofilm-associated infections, however, microbial persistence and metabolic cues from the wound microbiome can skew phagocyte polarization toward a dysfunctional state either sustaining a prolonged M1-like inflammatory profile that damages tissue or inducing an immunosuppressed M2-like state that allows microbial survival [42]

GGC attenuated the effect of oligomeric A 40 -induced changes in MMP-2 and MMP-9 activities in vitro

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